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"count": 732,
"next": "https://api.research.service.sci.tu.ac.th/api/normalPublication/?format=api&page=74",
"previous": "https://api.research.service.sci.tu.ac.th/api/normalPublication/?format=api&page=72",
"results": [
{
"id": 721,
"title": "Synthesis, structure, spectroscopy, and magnetism of two new dinuclear carbonato-bridged Cu(II) complexes",
"abstract": "Two new dinuclear μ-CO3 2- Cu(II) complexes with different coordination modes for the carbonato bridge have been obtained by fixation of atmospheric CO2 and also directly prepared from the carbonate salt. The compounds comprise: [Cu2(μ-CO3)(dpyam)4](ClO4) 2(H2O)4 (1), and [Cu2(μ-CO3)2(dpyam)2](H 2O) (2), (in which dpyam = di-2-pyridylamine). For 1, the carbonate ligand acts as a bridge between two Cu(II) centres showing an anti-anti (μ-η1-η1-CO3 2-) coordination mode with a distorted square-based pyramidal geometry for each Cu(II) environment. Complex 2 involves the di-μ-CO3 2- bridge with a novel tridentate μ-η1-η2-CO3 2- coordination mode. The geometry around each copper atom is distorted square-based pyramidal. Susceptibility measurements for both complexes show a weak to moderately strong antiferromagnetic coupling with J values of -90.4 and -9.9 cm-1 for 1 and 2, respectively. The tridentate co-ordination mode of the carbonate bridge in 2 has not previously been reported for dinuclear Cu(II) complexes. Also its magnetic behaviour and superexchange pathway are discussed. © 2001 Elsevier Science B.V. All rights reserved.",
"published_year": 2001,
"file": null,
"authors": [
"Kongsaeree P.",
"Youngme S.",
"Chaichit N.",
"Reedijk J.",
"Van Albada G.A."
],
"keywords": [
"carbonate chemistry",
"crystal structures",
"di-2-pyridylamine complexes",
"copper(ii) dimers",
"magnetism"
],
"references": [
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]
},
{
"id": 722,
"title": "Estimation of particulate matter from visibility in Bangkok, Thailand",
"abstract": "Lack of daily data on airborne particles has been a common problem in an air pollution research. To deal with this problem, a regression model was developed to estimate daily PM10 concentration using visibility in Bangkok from 1992 to 1997, based on 1092 visibility/PM10 pair-observations on low humidity days (humidity≤76.5%). Visibility was significantly and inversely associated with PM10 (r = 0.71), after adjusting for minimum temperature and winter indicator variable. The R2 of the model was 0.51.",
"published_year": 2001,
"file": null,
"authors": [
"Vajanapoom N.",
"Loomis D.",
"Neas L.M.",
"Shy C.M."
],
"keywords": [
"regression model",
"pm10",
"visibility",
"relative humidity",
"particulate matter"
],
"references": [
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"Malm, W.C., Theory of radiation transfer and visibility (1991) Acidic Deposition: State of Science and Technology. III, Terrestrial, Materials, Health and Visibility Effects, 3, pp. 25-41. , P.M. Irving (Eds.). The US National Acid Precipitation Assessment Program",
"Tsai, F.C., Smith, K.R., Vichit-Vadakan, N., Ostro, B.D., Chestnut, L.G., Kungskulniti, N., Indoor/outdoor PM10 and PM2.5 in Bangkok, Thailand (2000) J Exposure Anal Environ Epidemiol, 10, pp. 15-26",
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]
},
{
"id": 723,
"title": "Magnetic pair breaking by the Pr ions in the two `RE-123' EuBa2Cu3O7-δ and ErBa2Cu3O7-δ HTSCs codoped with Ca",
"abstract": "The suppression of superconductivity by the equal substitution of Pr and Ca into the Eu and Er `123' HTSCs are studied. The decreases in the Tcs of Eu1-2xPrxCaxBa2Cu3O7-δ and Er1-2xPrxCaxBa2Cu3O7-δ are observed to be linear as x increases from 0.0 to 0.010. The rate of suppression dTc/dx for the `Eu-(Pr,Ca)-123' HTCSs is larger than that for the `Er-(Pr,Ca)-123' HTSCs. It is observed that in the specimens studied, the Cu valencies (reflecting the hole concentration) for each of the doped specimen in the two series remain almost constant. This points to the suppression of superconductivity in the present `123' HTSCs by Pr substitution being due to magnetic pair breaking by the Pr ions. The relative magnitudes of the rates of suppression of the two systems and the of the Tcs of the host superconductors are taken to be manifestation of an rare earth size effect.",
"published_year": 2000,
"file": null,
"authors": [
"Varamit S.",
"Tang I.M.",
"Poomiput P."
],
"keywords": [],
"references": [
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]
},
{
"id": 724,
"title": "Pharmacokinetics of quinine in obesity",
"abstract": "Obesity can modify the pharmacokinetics of lipophilic drugs. As quinine is a lipophilic drug, this study was conducted to determine whether the pharmacokinetics of quinine is altered in obese subjects. Nine obese Thai men were compared with 8 age-matched lean men. After an oral dose of quinine had been given to the men, plasma quinine concentrations were measured up to 48 h after the dosing. Mean peak plasma quinine concentration in the obese group was significantly lower than that observed in the controls (4·0 ± 0·8 vs 5.0 ± 0·3 mg/L, P< 0·01). There were no significant differences in time to reach the peak plasma concentration, half-life and total clearance of quinine between the 2 groups. The mean clearances of quinine normalized to the ideal bodyweight (IBW) in the obese and the control groups were not significantly different (0·091 ± 0·018 vs 0·091 ± 0·024 L/h/kg IBW, P > 0·05). As there are similarities in the total clearance and the clearance of quinine based on IBW, the maintenance dose of quinine should be given to obese patients on the basis of ideal bodyweight, not on total bodyweight.",
"published_year": 2000,
"file": null,
"authors": [
"Viriyayudhakorn S.",
"Wanwimolruk S.",
"Ho P.C.",
"Thitiarchakul S.",
"Nachaisit S."
],
"keywords": [
"quinine",
"obesity",
"antimalarial drug",
"pharmacokinetics"
],
"references": [
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"Kuczmarski, R.J., Flegal, K.M., Campbell, S.M., Johnson, C.L., Increasing prevalence of overweight among US adults: The national health and nutrition examination surveys, 1960 to 1991 (1994) Journal of American Medical Association, 272, pp. 205-211",
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"Krishna, S., White, N.J., Pharmacokinetics of quinine, chloroquine and amodiaquine: Clinical implication (1996) Clinical Pharmacokinetics, 30, pp. 263-299",
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"Wanwimolruk, S., Kang, W., Coville, P.F., Viriyayudhakorn, S., Thitiarchakul, S., Marked enhancement by rifampicin and lack of effect of isoniazid on the elimination of quinine in man (1995) British Journal of Clinical Pharmacology, 40, pp. 87-91",
"Wanwimolruk, S., Wong, S.M., Coville, P.F., Viriyayudhakorn, S., Thitiarchakul, S., Cigarette smoking enhances the elimination of quinine (1993) British Journal of Clinical Pharmacology, 36, pp. 610-614",
"Abernethy, D.R., Greenblatt, D.J., Phenytoin disposition in obesity, determination of a loading dose (1985) Archives of Neurology, 42, pp. 468-471",
"Kotlyar, M., Carson, S.W., Effects of obesity on the cytochrome P450 enzyme system (1999) International Journal of Clinical Pharmacology and Therapeutics, 37, pp. 8-19",
"Babalola, C.P., Bolaji, O.O., Ogunbona, F.A., Sowunmi, A., Walker, O., Pharmacokinetics of quinine in African patients with acute falciparum malaria (1998) Pharmacy World and Science, 20, pp. 118-120",
"White, N.J., Antimalarial pharmacokinetics and treatment regimens (1992) British Journal of Clinical Pharmacology, 34, pp. 1-10"
]
},
{
"id": 725,
"title": "Acid-catalysed condensation of ethyl 5-acetoxymethyl-4-acetyl-3- methylpyrrole-2-carboxylate and its analogues with aromatic substrates",
"abstract": "The 5-acetoxy-4-acetylpyrrole 1 and its derivatives show a range of reactivity towards various aromatic substrates giving pyrroloindoles and pyrrolo[3,2-b]carbazoles amongst other products.",
"published_year": 1999,
"file": null,
"authors": [
"Chunchatprasert L.",
"Shannon P.V.R."
],
"keywords": [],
"references": [
"Chunchatprasert, L., Rao, K.R.N., Shannon, P.V.R., (1992) J. Chem. Soc., Perkin Trans. 1, p. 1779",
"Thyrann, T., Lightner, D.A., (1995) Tetrahedron Lett., 36, p. 4345",
"Fischer, H., Orth, H., (1934) Die Chemie des Pyrrols, 1. , Akademische Verlag, Leipzig",
"Chunchatprasert, L., Shannon, P.V.R., (1994) J. Chem. Soc., Perkin Trans. 1, p. 1765",
"Chunchatprasert, L., Shannon, P.V.R., (1996) J. Chem. Soc., Perkin Trans. 1, p. 1787",
"Clezy, P.S., Smythe, G.A., (1969) Aust. J. Chem., 22, p. 239"
]
},
{
"id": 726,
"title": "The effects of four mycotoxins on the mitogen stimulated proliferation of bovine peripheral blood mononuclear cells in vitro",
"abstract": "The effects of four mycotoxins, T-2 toxin, deoxynivalenol (DON), ochratoxin A (OTA) and fumonisin B1 (FB1) on the response of bovine peripheral blood mononuclear cells (PBM) in vitro to the mitogens concanavalin A (Con A), phytohaemagglutinin A (PHA) and pokeweed mitogen (PWM) were assayed after 4 days' incubation using 3H-tymidine uptake and the MTT bioassay. The concentrations of mycotoxin required to reduce the proliferative response of PBM by 50% for Con A, PHA and PWM as measured by 3H-thymidine incorporation was for T-2 toxin 0.30, 0.40 and 0.18 ng ml-1; for DON 0.07, 0.09 and 0.04 μg ml-1; for OTA 0.10, 0.20 and 0.15 μg ml-1, and for FB1 35, 18 and 11 μg ml-1 respectively. The concentrations of mycotoxin required to reduce the proliferative response of PBM by 50% for Con A, PHA and PWM as measured by the MTT bioassay were for T-2 toxin 2.0, 2.0 and 1.0 mg ml-1; for DON 0.70, 0.50 and 0.50 μg ml-1; OTA 1.5, 1.5 and 1.5 μg ml-1; and FB1 > 50, >50 and 20 μg ml-1 respectively. Further cytotoxicity assays including the LDH bioassay and Trypan blue exclusion were performed only on Con A-stimulated PBM cells after 72 h incubation. With the LDH-bioassay the 50% inhibition levels were T-2 toxin 0.3 ng ml-1, DON 0.4 μg ml-1, OTA 1.4 μg ml-1 and FB1 3.5 μg ml-1; for Trypan blue uptake the 50% inhibition levels were T-2 toxin 5 ng ml-1, DON 2.3 μg ml-1 and OTA 4 μg ml-1 respectively.",
"published_year": 1998,
"file": null,
"authors": [
"Smith J.E.",
"Fitzpatrick J.L.",
"Charoenpornsook K."
],
"keywords": [
"trypan blue exclusion",
"3h-thymidine uptake",
"ochratoxin a",
"bovine peripheral blood mononuclear cells",
"mtt and ldh bioassays",
"t-2 toxin",
"deoxynivalenol",
"fumonisin b1",
"cytotoxicity"
],
"references": [
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]
},
{
"id": 727,
"title": "Some investigations on the Bose-Srivastava algebra related to the multidimensional partially balanced association scheme",
"abstract": "We recall the multidimensional partially balanced (MDPB) association scheme defined by (, Sankhya, 26, 145-168). Here, we have k sets of objects, and association is defined within and between sets. The scheme within each set leads to the Bose-Mesner algebra, but the MDPB scheme as a whole leads to the (non-commutative) linear association algebra of Bose and Srivastava. We study the situation when k=2, and the two sets are identical, and furthermore, the schemes defined within sets are also identical. If the association between sets corresponds to an algebra which contains the algebra arising from association within sets, then the MDPB nature of the overall scheme is assured. But, we give examples to show that the relationship between sets may be more general, and yet an MDPB scheme may arise. We prove that this is true in general for arbitrary k.",
"published_year": 1998,
"file": null,
"authors": [
"Tungsarote R."
],
"keywords": [],
"references": [
"Bose, R.C., Srivastava, J.N., Multidimensional partially balanced designs and their analysis, with applications to partially balanced factorial fractions (1964) Sankhya Ser. a, 26, pp. 145-168"
]
},
{
"id": 728,
"title": "Clonning and sequence analysis of the 26 kDa glutathiones-tránsferase gene of schistosoma mekongi",
"abstract": "The number of genomic DNA or cDNA sequences of Schistosoma mekongi accessible in Genbank or EMBL is very limited up to now. Recently, two reports have appeared on the molecular phylogeny of Schistosoma species inferred from partial sequence data of rRNA genes; no further sequence data of S. mekongi is available yet. Knowledge of the molecular structure of protein coding genes of S. mekongi will provide a better understanding of gene function in the genus Schistosoma. A cDNA library of S. mekongi adult male was constructed and a cDNA encoding the 26 kDa glutathione S-transferase protein of this species was cloned. Sequence analysis of this cDNA confirmed the close phylogenetic relationship of S. mekongi to S. japonicum.",
"published_year": 1997,
"file": null,
"authors": [
"Korge G.",
"Vichasri-Grams S.",
"Grams R.",
"Viyanant V.",
"Upatham E.S."
],
"keywords": [],
"references": [
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"Barker, S.C., Blair, D., Molecularphylogeny of Schistosoma species supports traditional groupings within the genus (1996) J Parasitol, 82, pp. 292-298",
"Viyanant, V., Upatham, E.S., Isoenzyme analysis of Malaysian Schistosoma, S. mekongi and S. japonicum by isoelecfric focusing in polyacrylamide gel (1985) Southeast Asian J Trop Med Public Health, 16, pp. 539-545",
"Després, L., Imbert-Establet, D., Combes, C., Bonhomme, F., Molecular evidence linking hominid evolution to recent radiations of schistosomes (Platyhelminthes: Trematoda) (1992) Mol Phylogenet Evol, 1, pp. 295-304",
"Mannervik, B., The isoenzymes of glutathione-S-transferase (1985) Adv Enyzmol Relat Areas Mol Biol, 57, pp. 357-417",
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"Mozer, T.J., Tiemeier, D.C., Jaworski, E.G., Purification and characterization of corn glutathione S-transferase (1983) Biochemistry, 22, pp. 1068-1072",
"Capron, A., Dessaint, J.P., Capron, M., Pierce, R.J., Vaccine strategies against schistosomiasis (1992) Mem Inst Oswaldo Cruz, 87 (5 SUPPL.), pp. 1-9",
"O'Leary, K.A., Tracy, J.W., Purification of three cytosolic glutathione S-transferases from adult Schistosoma mansoni (1988) Arch Biochem Biopkys, 264, pp. 1-12"
]
},
{
"id": 729,
"title": "Pharmacokinetics of quinine in Asians (Thai) and white Caucasians",
"abstract": "The effect of ethnic factor on the pharmacokinetics of quinine was investigated by comparing its pharmacokinetic parameters in two different ethnic groups, 9 healthy Asian (Thai) and 10 white Caucasian subjects. After a single oral dose of quinine sulphate (600 mg) was given to the subjects, plasma quinine concentrations were measured by a specific high-performance liquid chromatography, up to 48 h after the dosing. Peak plasma quinine concentration and the time to peak concentration were similar in the Asian and Caucasian subjects. The mean apparent clearance of quinine in the Asians (0.095 ± 0.024 L/h/kg) was not significantly different from that in the Caucasian subjects (0.078 ± 0.02 L/h/hg). There was also no significant difference in the mean elimination half-life of quinine between the two groups. Lack of difference in the pharmacokinetics of quinine between the Asians and Caucasians suggests that the standard recommended doses of antimalarial quinine (600 mg quinine sulphate, 8 hourly) can be applied in both ethnic groups.",
"published_year": 1996,
"file": null,
"authors": [
"Viriyayudhakorn S.",
"Wanwimolruk S."
],
"keywords": [
"asians",
"pharmacokinetics",
"antimalarial drug",
"interethnic difference",
"quinine"
],
"references": [
"Zoest, A.R., Wanwimolruk, S., Hung, C.T., Simple high-performance liquid chromatographic method for the analysis of quinine in human plasma without extraction (1990) Journal of Liquid Chromatography, 13, pp. 3481-3491",
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"Wanwimolruk, S., Wong, S.M., Coville, P.F., Walker, R.J., In vitro hepatic microsomal metabolism of quinine: Identification of P450 enzymes responsible for quinine metabolism (1994) Canadian Journal of Physiology and Pharmacology, 72 (1 SUPPL.), p. 294",
"Kumana, C.R., Lauder, I.J., Chan, M., Ko, W., Lin, H.J., Differences in diazepam pharmacokinetics in Chinese and white Caucasians - Relation to body lipid stores (1987) European Journal of Clinical Pharmacology, 32, pp. 211-215",
"Fletcher, K.A., Price Evans, D.A., Gilles, H.M., Greaves, J., Bunnag, D., Harinasuta, T., Studies on the pharmacokinetics of primaquine (1981) Bulletin of the World Health Organization, 59, pp. 407-412",
"(1989) Weekly Epidemiological Record, 64, pp. 241-248",
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"Shimada, T., Yamazaki, H., Mimura, M., Inui, Y., Guengerich, F.P., Inter-individual variations in human liver cytochrome P-450 enzymes involved in the oxidation of drugs, carcinogens and toxic chemicals: Studies with liver microsomes of 30 Japanese and 30 Caucasians (1994) Journal of Pharmacology and Experimental Therapeutics, 270, pp. 414-423",
"Wernsdorfer, W.H., The development and spread of drug-resistant malaria (1991) Parasitology Today, 7, pp. 297-303",
"Wood, A.J.J., Zhou, H.H., Ethnic differences in drug disposition and responsiveness (1991) Clinical Pharmacokinetics, 20, pp. 350-373",
"Kalow, W., Ethnic differences in drug metabolism (1982) Clinical Pharmacokinetics, 7, pp. 373-400",
"Zhou, H.H., Koshakji, R.P., Silberstein, D.J., Wilkinson, G.R., Wood, A.J.J., Racial differences in drug response: Altered sensitivity and clearance of propranolol in men of Chinese descent as compared with American whites (1989) New England Journal of Medicine, 320, pp. 565-570",
"Wolff, P.H., Ethnic differences in alcohol sensitivity (1972) Science, 175, pp. 449-450",
"White, N.J., Clinical pharmacokinetics of antimalarial drugs (1985) Clinical Pharmacokinetics, 10, pp. 187-215",
"Wanwimolruk, S., Chalcroft, S., Lack of relationship between debrisoquine oxidation phenotype and the pharmacokinetics of quinine (1991) British Journal of Clinical Pharmacology, 32, pp. 617-620",
"Wanwimolruk, S., Wong, S.M., Coville, P.F., Viriyayudhakom, Thitiarchakul, S., Cigarette smoking enhances the elimination of quinine (1993) British Journal of Clinical Pharmacology, 36, pp. 610-614",
"Ghoniem, M.M., Korttila, K., Chiang, C.-K., Jacobs, L., Diazepam effects and kinetics in Caucasians and Orientals (1981) Clinical Pharmacology and Therapeutics, 29, pp. 749-756",
"Wanwimolruk, S., Kang, W., Coville, P.F., Viriyayudhakorn, S., Thitiarchakul, S., Marked enhancement by rifampicin and lack of effect of isoniazid on the elimination of quinine in man (1995) British Journal of Clinical Pharmacology, 40, pp. 87-91",
"White, N.J., Chanthavanich, P., Krishna, S., Bunch, C., Silamut, K., Quinine disposition kinetics (1983) British Journal of Clinical Pharmacology, 16, pp. 399-403",
"Wanwimolruk, S., Thou, M.R., Woods, D.J., Evidence for the polymorphic oxidation of debrisoquine and proguanil in a Khmer (Cambodian) population (1995) British Journal of Clinical Pharmacology, 40, pp. 166-169",
"Wanwimolruk, S., Sunbhanich, M., Pongmarutai, M., Patamasucon, P., Effects of cimetidine and ranitidine on the pharmacokinetics of quinine (1986) British Journal of Clinical Pharmacology, 22, pp. 346-350"
]
},
{
"id": 730,
"title": "Cigarette smoking enhances the elimination of quinine",
"abstract": "The pharmacokinetics of a single dose (600 mg) of quinine sulphate were examined in a group of non‐smokers (n = 10) and in heavy cigarette smokers (n = 10). The mean (± s.d.) oral clearance of quinine in smokers (0.189 ± 0.075 1 h−1 kg−1) was significantly greater than in non‐smokers (0.107 ± 0.0451 h−1 kg−1, P < 0.01). The unbound clearance of quinine which reflects activity of the drug‐metabolizing enzyme, was considerably greater (1.5‐fold) in the smokers than in the non‐smoker subjects. The mean elimination half‐life of quinine in smokers was 7.5 ± 1.4 (s.d.) h, significantly shorter (P < 0.005) than the mean value in non‐smokers (12.0 ± 3.1 h). These results suggest that cigarette smoking enhances the elimination of quinine. The clinical significance of these findings is unknown but they indicate the need for caution in the administration of quinine to patients who are heavy cigarette smokers. 1993 The British Pharmacological Society",
"published_year": 1993,
"file": null,
"authors": [
"COVILLE P.F.",
"THITIARCHAKUL S.",
"VIRIYAYUDHAKORN S.",
"WONG S.M.",
"WANWIMOLRUK S."
],
"keywords": [
"drug metabolism",
"enzyme induction",
"pharmacokinetics",
"smoking",
"quinine"
],
"references": [
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"Shader, RI, Greenblatt, DJ, Harmatz, JS, Franke, RI, Koch‐Weser, J., Absorption and disposition of chlordiazepoxide in young and elderly male volunteers (1977) J clin Pharmac, 17, pp. 709-715",
"Hart, P, Farrell, GC, Cooksley, WGE, Powell, LW., Enhanced drug metabolism in cigarette smokers (1976) Br med J, 2, pp. 147-149",
"Jusko, WJ, Schentag, JJ, Clark, JH, Gardner, M, Yurchak, AM., Enhanced biotransformation of theophylline in marijuana and tobacco smokers (1978) Clin Pharmac Ther, 24. , 460, 410",
"Küpfer, A, Branch, RA., Stereoselective mephenobarbital hydroxylation cosegregates with mephenytoin hydroxylation (1985) Clin Pharmac Ther, 38, pp. 414-418",
"Guengerich, FP, Martin, MV, Beaune, PH, Kremers, P, Wolff, T, Waxman, DJ., Characterization of the rat and human microsomal cytochrome P‐450 forms involved in nifedipine oxidation, a prototype for genetic polymorphism in oxidative drug metabolism (1986) J biol Chem, 261, pp. 5051-5056",
"Jusko, WJ., Role of tobacco smoking in pharmacokinetics (1978) J Pharmacokinet Biopharm, 6, pp. 7-39",
"Grygiel, JJ, Birkett, DJ., Cigarette smoking and theophylline clearance and metabolism (1981) Clin Pharmac Ther, 30, pp. 491-495",
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]
}
]
}